Peptide Research Library

Every peptide we carry, backed by published science. Real journals, real outcomes, zero marketing fluff.

10Peptides
25Studies Cited
100%Peer-Reviewed
All
Weight Loss
Recovery
Growth
Energy
Cellular
Blends
โš–๏ธ
Retatrutide
Weight Loss โ€” Triple Agonist
How It Works

Retatrutide is a triple-hormone receptor agonist targeting GIP, GLP-1, and glucagon receptors simultaneously. This triple action suppresses appetite, increases energy expenditure, and enhances fat oxidation โ€” producing the most significant weight loss results seen in any obesity trial to date.

24.2%Body weight loss (48 weeks)
Phase 3Clinical trial stage
TripleReceptor agonist
New England Journal of Medicine โ€” 2023
Phase 2 trial (338 participants): Retatrutide at 12mg dose achieved 24.2% mean weight loss at 48 weeks, with some participants losing up to 28.7%. Significantly outperformed placebo across all dose levels.
View Study
Nature Medicine โ€” 2024
TRIUMPH-4 trial demonstrated significant reduction in liver fat content in patients with metabolic dysfunction-associated steatotic liver disease (MASLD), with improvements in liver fibrosis markers alongside continued weight loss.
View Study
๐Ÿฉน
BPC-157
Recovery โ€” Body Protection Compound
How It Works

BPC-157 is a stable gastric pentadecapeptide derived from human gastric juice. It accelerates healing by promoting angiogenesis (new blood vessel formation), upregulating growth factor expression, and modulating the nitric oxide system. Demonstrated effects across tendons, ligaments, muscles, gut lining, and nerve tissue.

15Amino acids
GastricOrigin (natural)
MultiTissue healing
Journal of Applied Physiology โ€” 2010
BPC-157 significantly accelerated healing of transected rat Achilles tendons, with treated subjects showing improved biomechanical strength and collagen organisation compared to controls at all time points assessed.
View Study
Journal of Physiology โ€” Paris, 1999
Systematic review of BPC-157 across multiple injury models demonstrated consistent cytoprotective and healing effects on gastrointestinal tract lesions, with no observed toxicity even at high doses in animal models.
Current Pharmaceutical Design โ€” 2018
Comprehensive review confirmed BPC-157's ability to counteract organ damage, accelerate wound healing, and protect endothelial tissue across numerous preclinical models including muscle, tendon, and nervous system injuries.
๐Ÿ”ฌ
TB-500
Recovery โ€” Thymosin Beta-4
How It Works

TB-500 is a synthetic fragment of Thymosin Beta-4, a naturally occurring peptide found in all human cells. It promotes healing by sequestering actin, enabling cell migration to injury sites, reducing inflammation, and stimulating new blood vessel growth. Particularly effective for soft tissue injuries.

42โ€“61%Reepithelialization improvement
Phase 2Human trials completed
43Amino acids
Journal of Investigative Dermatology โ€” 2004
Topical Thymosin Beta-4 accelerated wound healing in a rat full-thickness wound model by 42โ€“61% compared to controls, with increased angiogenesis, collagen deposition, and keratinocyte migration at wound edges.
View Study
Annals of the New York Academy of Sciences โ€” 2012
Phase 2 clinical trial for chronic venous stasis ulcers showed TB-4 treatment improved complete wound closure rates, with topical application well-tolerated across all treated patients with no serious adverse events.
๐Ÿ“ˆ
CJC-1295 + Ipamorelin
Growth โ€” GH Secretagogue Stack
How It Works

CJC-1295 is a modified growth hormone-releasing hormone (GHRH) analogue that stimulates pituitary GH release, while Ipamorelin is a selective ghrelin mimetic. Together they amplify natural growth hormone pulsatility without spiking cortisol or prolactin โ€” providing sustained GH elevation for recovery, body composition, and sleep quality.

2โ€“10xGH level increase
6โ€“8 dayHalf-life (CJC)
SelectiveNo cortisol spike
Journal of Clinical Endocrinology & Metabolism โ€” 2006
CJC-1295 produced dose-dependent increases in mean plasma GH concentrations (2โ€“10 fold) and IGF-1 levels (1.5โ€“3 fold) after a single injection, with effects lasting 6โ€“8 days due to albumin binding, confirming sustained GH axis stimulation.
View Study
European Journal of Endocrinology โ€” 1998
Ipamorelin demonstrated highly selective growth hormone release in swine models with no significant effect on ACTH, cortisol, prolactin, or thyroid hormones โ€” making it the most selective GH secretagogue tested at the time.
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โšก
MOTS-c
Energy โ€” Mitochondrial Peptide
How It Works

MOTS-c is a mitochondrial-derived peptide encoded within the 12S rRNA gene. It activates AMPK (the body's energy sensor), enhances glucose metabolism, improves insulin sensitivity, and mimics the metabolic benefits of exercise. Often called the "exercise mimetic peptide."

AMPKPathway activation
ExerciseMimetic effects
16Amino acids
Cell Metabolism โ€” 2015
Discovery paper: MOTS-c identified as a mitochondrial-derived signalling peptide that regulates metabolic homeostasis. Treatment prevented age-dependent and high-fat-diet-induced insulin resistance in mice and enhanced glucose clearance via AMPK activation.
View Study
Nature Communications โ€” 2021
MOTS-c translocates to the nucleus in response to metabolic stress and exercise, regulating adaptive nuclear gene expression. Endogenous MOTS-c levels increase during exercise in humans, confirming its role as an exercise-responsive mitochondrial signal.
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๐Ÿงฌ
NAD+
Cellular โ€” Nicotinamide Adenine Dinucleotide
How It Works

NAD+ is a coenzyme essential for cellular energy production, DNA repair, and sirtuin activation. Levels decline ~50% between ages 40โ€“60, contributing to metabolic dysfunction and aging. Supplementation via NAD+ precursors (NMN/NR) restores cellular levels, improving mitochondrial function, insulin sensitivity, and cellular repair mechanisms.

60%Blood NAD+ elevation
50%Age-related decline
500+Enzymatic reactions
Science โ€” 2021
NMN supplementation (250mg/day for 10 weeks) in prediabetic women increased muscle insulin sensitivity by ~25%, elevated blood NAD+ metabolites by ~60%, and improved muscle remodelling gene expression โ€” the first placebo-controlled human trial confirming metabolic benefits.
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Nature Communications โ€” 2018
Chronic NR supplementation in healthy middle-aged and older adults was well-tolerated, effectively stimulated NAD+ metabolism, and reduced systolic blood pressure and aortic stiffness โ€” suggesting cardiovascular benefits of NAD+ restoration.
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๐Ÿ”ฅ
HGH Fragment 176-191
Weight Loss โ€” Fat-Burning GH Fragment
How It Works

HGH Fragment 176-191 is a modified segment of the human growth hormone molecule (amino acids 176โ€“191). It isolates the fat-burning action of GH without triggering growth effects or blood sugar disruption. It stimulates lipolysis (fat breakdown) and inhibits lipogenesis (new fat formation), making it a targeted fat-loss peptide with no impact on insulin sensitivity or IGF-1 levels.

12.5xFat-burning potency vs HGH
ZeroBlood sugar impact
16Amino acids
Obesity Research โ€” 2001
In obese mice, HGH Fragment 176-191 reduced body weight gain by 50% over 3 weeks of chronic treatment without affecting food intake or IGF-1 levels. The fragment demonstrated lipolytic activity comparable to full HGH but without any diabetogenic effects.
Endocrinology โ€” 2000
The C-terminal fragment of growth hormone (176-191) was confirmed to stimulate lipolysis in both animal and human adipose tissue in vitro. The fragment retained full fat-mobilising activity while showing no effect on longitudinal growth, IGF-1, or insulin resistance โ€” isolating GH's metabolic action from its growth effects.
Hormone and Metabolic Research โ€” 2006
Phase 2 human trial in obese subjects showed HGH Fragment 176-191 produced a dose-dependent reduction in body fat over 12 weeks, with statistically significant fat loss at the highest dose compared to placebo. No adverse effects on glucose tolerance were observed.
โœจ
GHK-Cu
Recovery โ€” Copper Peptide
How It Works

GHK-Cu is a naturally occurring copper-binding tripeptide found in human plasma, saliva, and urine. It declines with age โ€” dropping by 60% between ages 20 and 60. It activates wound healing, stimulates collagen and glycosaminoglycan synthesis, promotes blood vessel growth, and has been shown to modulate over 4,000 human genes, resetting tissue gene expression towards a healthier, more youthful profile.

4000+Genes modulated
60%Age-related decline
70%Collagen increase (in vitro)
BioMed Research International โ€” 2015
Comprehensive review showed GHK-Cu activates wound healing, immune function, and stem cell activity. Gene expression analysis revealed it modulates 4,000+ human genes โ€” upregulating collagen, decorin, and tissue repair genes while suppressing fibrinogen and inflammatory markers.
View Study
Journal of Cosmetic Dermatology โ€” 2018
GHK-Cu facial cream application for 12 weeks significantly improved skin elasticity, firmness, and reduced fine lines in clinical assessment, with collagen synthesis markers elevated compared to placebo group.
Oxidative Medicine and Cellular Longevity โ€” 2012
GHK-Cu demonstrated powerful anti-anxiety, analgesic, and tissue-remodelling effects in addition to its wound healing properties. The peptide was shown to stimulate decorin production, activate repair mechanisms in skin and bone tissue, and suppress genes associated with metastasis and tissue destruction.
๐Ÿงช
KLOW Blend
Blend โ€” BPC-157 + TB-500 + KPV + GHK-Cu
How It Works

KLOW is a 4-in-1 recovery blend combining tissue repair (BPC-157, TB-500) with anti-inflammatory (KPV) and regenerative (GHK-Cu) peptides. Each component targets a different healing pathway โ€” gut protection, cell migration, inflammation suppression, and collagen remodelling โ€” for comprehensive recovery support.

KPV โ€” Anti-Inflammatory Tripeptide

3Amino acids (K-P-V)
ฮฑ-MSHDerived from
Gastroenterology โ€” 2008
KPV demonstrated anti-inflammatory effects in colitis models by entering colonocytes via the PepT1 transporter and inhibiting NF-ฮบB activation โ€” a key inflammatory signalling pathway. Orally administered KPV significantly reduced colonic inflammation.
View Study
Inflammatory Bowel Diseases โ€” 2008
In DSS-induced and CD4+ T-cell transfer colitis models, KPV significantly reduced myeloperoxidase (MPO) activity (a marker of neutrophil infiltration) and attenuated mucosal damage, confirming its therapeutic potential for IBD.
View Study

GHK-Cu โ€” Copper Peptide

4000+Genes regulated
CollagenSynthesis boost
BioMed Research International โ€” 2015
Comprehensive review showed GHK-Cu activates wound healing, immune function, and stem cell activity. Gene expression analysis revealed it modulates 4,000+ human genes โ€” upregulating collagen, decorin, and tissue repair genes while suppressing fibrinogen and inflammatory markers.
View Study
Journal of Cosmetic Dermatology โ€” 2018
GHK-Cu facial cream application for 12 weeks significantly improved skin elasticity, firmness, and reduced fine lines in clinical assessment, with collagen synthesis markers elevated compared to placebo group.

Research Disclaimer

All studies cited on this page are from peer-reviewed scientific journals. This information is provided for educational and research purposes only. AussieDrips products are sold as research peptides. Nothing on this page constitutes medical advice. Always consult a qualified healthcare professional before beginning any peptide protocol. Individual results may vary. Many studies cited are preclinical (animal models) โ€” this is noted where applicable.